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Clinical Medical
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ISSN: 2474-3682
IMAGE ARTICLE | VOLUME 2, ISSUE 1 | OPEN ACCESS DOI: 10.23937/2474-3682/1510026

A Different Form of Acute Myocardial Infarction!

Rita Ferreira

Serviço de Cardiologia A, Centro Hospitalar e Universitário de Coimbra, Portugal

*Corresponding author: Ana Rita Ferreira, cardiology resident, Serviço de Cardiologia A, Centro Hospitalar e Universitário de Coimbra, Coimbra, Portugal, E-mail: ritafmup@gmail.com

Published: January 30, 2016

Citation: Ferreira R (2016) A Different Form of Acute Myocardial Infarction!. Clin Med Img Lib 2:026. doi.org/10.23937/2474-3682/1510026

Copyright: © 2016 Ferreira R. This is an open-access content distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.


Keywords


Acute coronary syndrome, Complete left bundle branch block, Endocarditis

A 50-year old man was admitted to the emergency room due to severe dyspnea and thoracic pain with 3 hours of onset. He had no known history of cardiovascular disease, but he was recovering from drug addict. At the emergency room he was at cardiogenic shock requiring intubation and mechanical ventilation.

Physical exam was notable for diffuse lungs crackles. The heart sounds were normal and a grade III/VI systolic murmur was audible. The 12-lead electrocardiogram demonstrated sinus tachycardia and new complete left bundle branch block (Figure I).

The diagnosis of acute myocardial infarction with ST-segment elevation was done. The interventional cardiologist was contacted to perform primary angioplasty. The clinical cardiologist was also called, and during the waiting time for hemodynamic team he performed a transthoracic echocardiography. That showed a enlargement of left ventricle, with mild impaired function and a large mass in the ventricular side of the aortic valve, conditioning obstruction and major aortic valve insufficiency (Figure II). A transesophageal echocardiography was also perfumed and confirm the aortic valve destruction and a large paravalvular abscess involving the left sinus of Valsalva (Figure II and Figure III). Blood cultures were collected and empirical antibiotic therapy for endocarditis was initiated.

Coronary angiography was not performed due to the high risk of this procedure.

The case was discussed with the cardiac surgeon but they refused the patient and he died a few hours later.